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Publications

MHC class II antigen presentation by intestinal epithelial cells fine-tunes bacteria-reactive CD4 T-cell responses.

Heuberger CE. et al, (2024), Mucosal Immunol, 17, 416 - 430

Adipocyte autophagy limits gut inflammation by controlling oxylipin and IL-10.

Richter FC. et al, (2023), EMBO J

MHC class II antigen presentation by intestinal epithelial cells fine-tunes bacteria-reactive CD4 T cell responses

Heuberger CE. et al, (2023)

Cross-tissue, single-cell stromal atlas identifies shared pathological fibroblast phenotypes in four chronic inflammatory diseases.

Korsunsky I. et al, (2022), Med (N Y), 3, 481 - 518.e14

IL-1-driven stromal-neutrophil interactions define a subset of patients with inflammatory bowel disease that does not respond to therapies.

Friedrich M. et al, (2021), Nat Med, 27, 1970 - 1981

Adipocyte autophagy limits gut inflammation by controlling oxylipin levels

Richter FC. et al, (2021)

Chronic Alcohol Consumption Exacerbates the Severity of Psoriasiform Dermatitis in Mice.

Vasseur P. et al, (2020), Alcoholism, clinical and experimental research, 44, 1728 - 1733

Cytokine Networks in the Pathophysiology of Inflammatory Bowel Disease.

Friedrich M. et al, (2019), Immunity, 50, 992 - 1006

Liver fibrosis is associated with cutaneous inflammation in the imiquimod-induced murine model of psoriasiform dermatitis.

Vasseur P. et al, (2018), The British journal of dermatology, 179, 101 - 109

Development of a new model of reconstituted mouse epidermis and characterization of its response to proinflammatory cytokines.

Pohin M. et al, (2018), Journal of tissue engineering and regenerative medicine, 12, e1098 - e1107

Interleukin-17A-induced production of acute serum amyloid A by keratinocytes contributes to psoriasis pathogenesis.

Couderc E. et al, (2017), PloS one, 12

High-Fat Diet-Induced IL-17A Exacerbates Psoriasiform Dermatitis in a Mouse Model of Steatohepatitis.

Vasseur P. et al, (2016), The American journal of pathology, 186, 2292 - 2301

Oncostatin M overexpression induces skin inflammation but is not required in the mouse model of imiquimod-induced psoriasis-like inflammation.

Pohin M. et al, (2016), European journal of immunology, 46, 1737 - 1751

IMQ-induced skin inflammation in mice is dependent on IL-1R1 and MyD88 signaling but independent of the NLRP3 inflammasome.

Rabeony H. et al, (2015), European journal of immunology, 45, 2847 - 2857

Include forthcoming?