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Publications

MHC class II antigen presentation by intestinal epithelial cells fine-tunes bacteria-reactive CD4 T-cell responses.

Heuberger CE. et al, (2024), Mucosal Immunol, 17, 416 - 430

Elevated type-17 cytokines are present in Axial Spondyloarthritis stool

BOWNESS P. and BROUGH I., (2024), Discovery Immunology

Blimp-1 and c-Maf regulate immune gene networks to protect against distinct pathways of pathobiont-induced colitis.

Alvarez-Martinez M. et al, (2024), Nat Immunol

Immune microniches shape intestinal Treg function.

Gu Y. et al, (2024), Nature

Alterations in the gut microbiome implicate key taxa and metabolic pathways across inflammatory arthritis phenotypes.

Thompson KN. et al, (2023), Sci Transl Med, 15

Adipocyte autophagy limits gut inflammation by controlling oxylipin and IL-10.

Richter FC. et al, (2023), EMBO J

MHC class II antigen presentation by intestinal epithelial cells fine-tunes bacteria-reactive CD4 T cell responses

Heuberger CE. et al, (2023)

Fates of Commensal Specific, Innate-like CD8+T cells in the Gut

Farley TK. et al, (2023), JOURNAL OF IMMUNOLOGY, 210

A conserved population of MHC II-restricted, innate-like, commensal-reactive T cells in the gut of humans and mice.

Hackstein C-P. et al, (2022), Nat Commun, 13

A viral cocktail calms gut inflammation.

Bertocchi A. and Powrie F., (2022), Nature, 612, 220 - 221

Anti-TNF-induced IL-27 modulates regulatory T cell responses in patients with IBD that respond to therapy

Peres RS. et al, (2022)

Hermansky-Pudlak syndrome type 1 causes impaired anti-microbial immunity and inflammation due to dysregulated immunometabolism.

Cavounidis A. et al, (2022), Mucosal Immunol

A conserved population of MHC II-restricted, innate-like, commensal-reactive T cells in the gut of humans and mice

Hackstein C-P. et al, (2022)

Cross-tissue, single-cell stromal atlas identifies shared pathological fibroblast phenotypes in four chronic inflammatory diseases.

Korsunsky I. et al, (2022), Med (N Y), 3, 481 - 518.e14

Author Correction: Somatic mosaicism and common genetic variation contribute to the risk of very-early-onset inflammatory bowel disease.

Serra EG. et al, (2022), Nat Commun, 13

Genetic and environmental factors shape the host response to Helicobacter hepaticus: insights into IBD pathogenesis.

Jeffery R. et al, (2022), Curr Opin Microbiol, 65, 145 - 155

Intestinal lamina propria supports acquired eTreg suppressor function

Gu Y. et al, (2022)

IL-1-driven stromal-neutrophil interactions define a subset of patients with inflammatory bowel disease that does not respond to therapies.

Friedrich M. et al, (2021), Nat Med, 27, 1970 - 1981

Adipocyte autophagy limits gut inflammation by controlling oxylipin levels

Richter FC. et al, (2021)

Tissue-dependent transcriptional and bacterial associations in primary sclerosing cholangitis-associated inflammatory bowel disease

Ilott NE. et al, (2021), Wellcome Open Research, 6, 199 - 199

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