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Immunomodulation By a Second-Generation Peptidyl Arginine Deiminase Inhibitor Abrogates Collagen-Induced Arthritis in a Therapeutic Protocol
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Bronchiectasis is a Model for Chronic Bacterial Infection Inducing Autoimmunity in Rheumatoid Arthritis.
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Expression of citrulline and homocitrulline residues in the lungs of non-smokers and smokers: implications for autoimmunity in rheumatoid arthritis.
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PPAD remains a credible candidate for inducing autoimmunity in rheumatoid arthritis: comment on the article by Konig et al.
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SMOKING AFFECTS IMMUNE RESPONSE TO CITRULLINATED AUTOANTIGENS BEFORE CLINICAL ONSET OF RHEUMATOID ARTHRITIS IN A SOUTHERN EUROPEAN COHORT
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A1.5 Smoking is a risk factor for ACPA prior to onset of symptoms of rheumatoid arthritis in a cohort from southern europe.
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The autoantibody repertoire in periodontitis: a role in the induction of autoimmunity to citrullinated proteins in rheumatoid arthritis?
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Heightened immune response to autocitrullinated Porphyromonas gingivalis peptidylarginine deiminase: A potential mechanism for breaching immunologic tolerance in rheumatoid arthritis
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Porphyromonas gingivalis facilitates the development and progression of destructive arthritis through its unique bacterial peptidylarginine deiminase (PAD).
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IS PERIODONTAL DISEASE A RISK FACTOR FOR RHEUMATOID ARTHRITIS? THE ANTI-CITRULLINATED ANTIBODY REPERTOIRE IN PERIODONTAL DISEASE
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