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Whether resident and recruited myeloid cells may impair or aid healing of acute skin wounds remains a debated question. To begin to address this, we examined the importance of CD11c+ myeloid cells in the early activation of skin wound repair. We find that an absence of CD11c+ cells delays wound closure and epidermal proliferation, likely due to defects in the activation of the IL-23-IL-22 axis that is required for wound healing.

Original publication

DOI

10.1111/imm.13312

Type

Journal article

Journal

Immunology

Publication Date

05/2021

Volume

163

Pages

105 - 111

Keywords

CD11c, Langerhans cells, dendritic cells, skin, wound repair, Animals, CD11 Antigens, Dendritic Cells, Disease Models, Animal, Kinetics, Langerhans Cells, Mice, Inbred C57BL, Mice, Transgenic, Skin, Wound Healing, Wounds and Injuries, Mice