Thibault Griseri

PhD

University Research Lecturer and Arthritis Research UK Career Development Fellow

After a PhD in Immunology in France working on NKT cells and type 1 diabetes, I moved to the University of Oxford in 2007 to work as a postdoctoral researcher in Prof. Fiona Powrie's laboratory, investigating the regulation of immune response in chronic intestinal inflammation.

In 2013 I joined the Kennedy Institute of Rheumatology to work on chronic inflammatory arthritis with Prof Sir Marc Feldmann and subsequently obtained a career development fellowship funded by Arthritis Research UK.

ILC3 GM-CSF production and mobilisation orchestrate acute intestinal inflammation.
Pearson C. et al, (2016), Elife, 5
Granulocyte Macrophage Colony-Stimulating Factor-Activated Eosinophils Promote Interleukin-23 Driven Chronic Colitis.
Griseri T. et al, (2015), Immunity, 43, 187 - 199
The alarmin IL-33 promotes regulatory T-cell function in the intestine.
Schiering C. et al, (2014), Nature, 513, 564 - 568
CTLA-4 promotes Foxp3 induction and regulatory T cell accumulation in the intestinal lamina propria.
Barnes MJ. et al, (2013), Mucosal Immunol, 6, 324 - 334
Dysregulated Hematopoietic Stem and Progenitor Cell Activity Promotes Interleukin-23-Driven Chronic Intestinal Inflammation
Griseri T. et al, (2012), IMMUNITY, 37, 1116 - 1129
OX40 is required for regulatory T cell-mediated control of colitis.
Griseri T. et al, (2010), J Exp Med, 207, 699 - 709
NKT cell-plasmacytoid dendritic cell cooperation via OX40 controls viral infection in a tissue-specific manner.
Diana J. et al, (2009), Immunity, 30, 289 - 299
Prevention of type 1 diabetes by invariant NKT cells is independent of peripheral CD1d expression.
Novak J. et al, (2007), J Immunol, 178, 1332 - 1340
Regulation of type 1 diabetes by NKT cells.
Novak J. et al, (2007), Int Rev Immunol, 26, 49 - 72
The INKT cells prevent diabetes type 1 blocking viral replication in pancreas.
Griseri T. et al, (2006), DIABETES & METABOLISM, 32, S39 - S39
Invariant NKT cells exacerbate type 1 diabetes induced by CD8 T cells.
Griseri T. et al, (2005), J Immunol, 175, 2091 - 2101
Inhibition of T cell differentiation into effectors by NKT cells requires cell contacts.
Novak J. et al, (2005), J Immunol, 174, 1954 - 1961

Key publications

Granulocyte Macrophage Colony-Stimulating Factor-Activated Eosinophils Promote Interleukin-23 Driven Chronic Colitis.
Griseri T. et al, (2015), Immunity, 43, 187 - 199
The alarmin IL-33 promotes regulatory T-cell function in the intestine.
Schiering C. et al, (2014), Nature, 513, 564 - 568
Dysregulated Hematopoietic Stem and Progenitor Cell Activity Promotes Interleukin-23-Driven Chronic Intestinal Inflammation
Griseri T. et al, (2012), IMMUNITY, 37, 1116 - 1129
OX40 is required for regulatory T cell-mediated control of colitis.
Griseri T. et al, (2010), J Exp Med, 207, 699 - 709
NKT cell-plasmacytoid dendritic cell cooperation via OX40 controls viral infection in a tissue-specific manner.
Diana J. et al, (2009), Immunity, 30, 289 - 299

Recent Publications

ILC3 GM-CSF production and mobilisation orchestrate acute intestinal inflammation.
Pearson C. et al, (2016), Elife, 5
Granulocyte Macrophage Colony-Stimulating Factor-Activated Eosinophils Promote Interleukin-23 Driven Chronic Colitis.
Griseri T. et al, (2015), Immunity, 43, 187 - 199
The alarmin IL-33 promotes regulatory T-cell function in the intestine.
Schiering C. et al, (2014), Nature, 513, 564 - 568
CTLA-4 promotes Foxp3 induction and regulatory T cell accumulation in the intestinal lamina propria.
Barnes MJ. et al, (2013), Mucosal Immunol, 6, 324 - 334
Dysregulated Hematopoietic Stem and Progenitor Cell Activity Promotes Interleukin-23-Driven Chronic Intestinal Inflammation
Griseri T. et al, (2012), IMMUNITY, 37, 1116 - 1129

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